Difference between revisions of "YlaC"
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* '''Effectors of protein activity:''' YlaD acts as anti-sigma {{PubMed|16728958}} | * '''Effectors of protein activity:''' YlaD acts as anti-sigma {{PubMed|16728958}} | ||
| − | * '''Interactions:''' [[YlaC]]-[[YlaD]] {{PubMed|16728958}}, [[YlaC]]-[[RpoB]]-[[RpoC]] | + | * '''[[SubtInteract|Interactions]]:''' |
| + | ** [[YlaC]]-[[YlaD]] {{PubMed|16728958}}, [[YlaC]]-[[RpoB]]-[[RpoC]] | ||
| − | * '''Localization:''' | + | * '''[[Localization]]:''' |
=== Database entries === | === Database entries === | ||
Revision as of 09:28, 2 November 2011
- Description: RNA polymerase sigma factor YlaC
| Gene name | ylaC |
| Synonyms | |
| Essential | no |
| Product | RNA polymerase sigma factor YlaC |
| Function | transcription |
| Interactions involving this protein in SubtInteract: YlaC | |
| MW, pI | 20 kDa, 9.282 |
| Gene length, protein length | 519 bp, 173 aa |
| Immediate neighbours | ylaB, ylaD |
| Get the DNA and protein sequences (Barbe et al., 2009) | |
Genetic context
This image was kindly provided by SubtiList
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Contents
Categories containing this gene/protein
transcription, sigma factors and their control
This gene is a member of the following regulons
The YlaC regulon: ylaA-ylaB-ylaC-ylaD
The gene
Basic information
- Locus tag: BSU14730
Phenotypes of a mutant
Database entries
- DBTBS entry: [1]
- SubtiList entry: [2]
Additional information
The protein
Basic information/ Evolution
- Catalyzed reaction/ biological activity:
- Protein family: ECF subfamily (according to Swiss-Prot)
- Paralogous protein(s):
Extended information on the protein
- Kinetic information:
- Domains:
- Modification:
- Cofactor(s):
- Effectors of protein activity: YlaD acts as anti-sigma PubMed
Database entries
- Structure:
- UniProt: O07627
- KEGG entry: [3]
- E.C. number:
Additional information
Expression and regulation
- Regulation:
- Additional information:
Biological materials
- Mutant:
- Expression vector:
- lacZ fusion:
- GFP fusion:
- two-hybrid system:
- Antibody:
Labs working on this gene/protein
Your additional remarks
References
Additional publications: PubMed
